Research provenance
This page is the master public research library for named BioAtlas papers, Zenodo records, preprints, reports, and DOI-backed provenance. The grouped families show how each paper supports the wider public pages: physics-first biology, QTOS, Rubik, ECS, oncology, microbiome, cell atlas, systems biology, safety, licensing, and diligence.
Named DOI records
46
Research families
10
Public role
Provenance
Protected depth
Reviewed access
BioAtlas public estate map
BioAtlas is staged deliberately: public proof, research provenance, platform engines, commercial packaging, protected diligence, and reviewed access. Each layer shows enough for the right audience without exposing protected routes, data-room material, or internal app surfaces.
Readable public pages, safe summaries, route maps, DOI links, and the high-level BioAtlas story.
DOI-backed papers, preprints, Zenodo records, grouped research collections, and public provenance.
QTOS, Rubik, ECS, oncology, microbiome, cell atlas, systems biology, neuro, and infrastructure families.
Licensing families, dataset access, SaaS/API routes, white-label scope, and AI-rights boundaries.
Buyer review, data-room categories, staged release, provenance, IP boundaries, and technical diligence.
Serious research, licensing, diligence, platform, API, and acquisition routes start with reviewed access.
Research families
Use this section as the public map. Each family links the papers to the relevant public page stories without exposing protected vault material or private data-room files.
The foundation layer: collapse dynamics, QTOS, Rubik, constraint-first computation, irreversibility, reachability, and biological state limits.
6 records
The ECS layer: endocannabinoid integrity, immunology, ageing, mitochondrial bioenergetics, systems-regulated operating-model logic, and biological coordination.
5 records
A context-dependent series separating THC exposure from ECS medicine and framing cannabinoid risk through allostasis, adaptive capacity, and indication-aware use.
7 records
Cancer as systems failure: hallmarks, PCD coordination, ECS dysregulation, tumour microenvironment, immune evasion, metabolism, and persistent signalling.
11 records
The gut as a signalling interface: microbiome, immunity, mitochondria, ECS tone, neuro-axis regulation, and allostatic state.
7 records
Neural and sensory systems: neuroplasticity, epilepsy, pain, sleep, cognition, ECS modulation, and nervous-system state transitions.
6 records
Immune-system coordination through inflammation, surveillance, mitochondrial bioenergetics, systems immunology, and inflammatory-system context.
6 records
Metabolic and adaptive capacity records linking allostasis, mitochondria, ageing, menopause, fatigue context, metabolic reprogramming, and state-based modelling.
6 records
The exploratory systems biology layer: DNA, proton tunnelling, biophotonics, bioelectromagnetic interfaces, and speculative physics boundaries.
5 records
Safety and governance records for non-claims, identity-threat, AI-use boundaries, irreversibility, constraints, and controlled public access.
6 records
Research family
The foundation layer: collapse dynamics, QTOS, Rubik, constraint-first computation, irreversibility, reachability, and biological state limits.
Introduces the physics-first disease-dynamics framework across layered collapse, coherence, constraints, and state transitions.
Early QTOS record describing the systems-state operating-model concept and platform-level architecture.
Defines the QTOS capability envelope, limits, guardrails, non-claims, and infrastructure-grade scope.
Defines Rubik as a constraint-first computation and governance layer below QTOS, focused on reachability, impossibility, and deterministic boundaries.
Distinguishes descriptive mapping from constraint systems that define what is reachable, blocked, or structurally impossible.
Frames irreversibility, intervention limits, and biological state governance as a safety and systems problem.
Research family
The ECS layer: endocannabinoid integrity, immunology, ageing, mitochondrial bioenergetics, systems-regulated operating-model logic, and biological coordination.
Frames the ECS as a systems-regulated operating-model layer across biological coordination, state interpretation, and systems integration.
Explores ECS involvement in inflammation, autoimmunity, immune surveillance, and immune-system coordination.
Connects ECS with mitochondrial bioenergetics, ATP generation, redox control, fatigue context, and system resilience.
Connects ECS signalling with ageing, hormone context, longevity, allostasis, and adaptive capacity.
Umbrella record for cannabinoid risk, endocannabinoid integrity, allostasis, adaptive capacity, and indication-aware frameworks.
Research family
A context-dependent series separating THC exposure from ECS medicine and framing cannabinoid risk through allostasis, adaptive capacity, and indication-aware use.
Introduces the need for indication-aware cannabinoid framing rather than generic THC-positive or THC-negative claims.
Frames chronic THC exposure through metabolic allostasis, glycaemic dysregulation, and context-dependent biological risk.
Explores autonomic and cardiovascular allostatic load under sustained CB1 signalling.
Connects sustained CB1-dominant signalling with allostatic immune suppression and cancer-surveillance failure risk.
Positions THC as a regulatory modulator whose effects depend on biological context, indication, resilience, and system state.
Separates THC exposure from broader endocannabinoid medicine and argues for indication-aware, context-dependent ECS interpretation.
Presents indication-aware cannabinoid framing for separating context, risk, adaptive capacity, and ECS integrity.
Research family
Cancer as systems failure: hallmarks, PCD coordination, ECS dysregulation, tumour microenvironment, immune evasion, metabolism, and persistent signalling.
Maps 22 cancer hallmarks against ECS, signalling, and systems-biology layers for oncology intelligence.
Connects ECS modulation with programmed cell-death classes and cell-fate systems interpretation.
A systems oncology research record around systems-level cancer interpretation and AI-supported oncology intelligence.
Frames metabolic reprogramming as a core systems-biology and oncology intelligence layer.
An oncology research record connecting OV context, nutrigenetics, microbiome, frequency, and enzyme-driven systems framing.
Research provenance record connecting GcMAF, Nagalase suppression, fibrin context, macrophage activation discussion, immune signalling, and systems-biology review boundaries.
Connects ECS signalling with tumour microenvironment behaviour, immune evasion, and apoptosis context.
Positions ECS dysregulation as a systems-level failure of biological coordination across cancer hallmarks.
Links cancer hallmark logic with programmed cell-death coordination and endocannabinoid signalling.
Frames persistent signalling and drug resistance as a compartmental redistribution problem rather than a single-pathway failure.
Connects proton tunnelling, base-pair instability, epigenetic collapse, and oncogenesis as a systems biology research thread.
Research family
The gut as a signalling interface: microbiome, immunity, mitochondria, ECS tone, neuro-axis regulation, and allostatic state.
Integrates ECS, microbiome, gut-immune-neuro signalling, systems biology, and systems-biology context.
Frames the ECS as a systems-regulated operating-model layer across biological coordination, state interpretation, and systems integration.
Connects ECS with mitochondrial bioenergetics, ATP generation, redox control, fatigue context, and system resilience.
Explores ECS involvement in inflammation, autoimmunity, immune surveillance, and immune-system coordination.
Research provenance record connecting GcMAF, Nagalase suppression, fibrin context, macrophage activation discussion, immune signalling, and systems-biology review boundaries.
Defines a programmable operating-system framing for neural, cognitive, gut-brain, and ECS reprogramming research.
Umbrella record for cannabinoid risk, endocannabinoid integrity, allostasis, adaptive capacity, and indication-aware frameworks.
Research family
Neural and sensory systems: neuroplasticity, epilepsy, pain, sleep, cognition, ECS modulation, and nervous-system state transitions.
Defines a programmable operating-system framing for neural, cognitive, gut-brain, and ECS reprogramming research.
A neuro-systems-state integration dataset and framework for neural cells, cell-state interpretation, and systems modelling.
Connects nervous-system reprogramming, neural epigenetics, resonance tuning, and ECS-modulated repair context.
Extends miRNA biointelligence into neuroplasticity, neural repair, and nervous-system systems logic.
Frames epilepsy through mitochondrial dysfunction, signalling modulation, systems dynamics, and AI-assisted interpretation.
Explores ECS, sleep, sensory regulation, signalling biology, and neuroimmune framing.
Research family
Immune-system coordination through inflammation, surveillance, mitochondrial bioenergetics, systems immunology, and inflammatory-system context.
Frames immune-system bioenergetics through systems immunology, AI-assisted interpretation, and immune recalibration context.
A broad immunology and systems-bioenergetics record connecting immune recalibration, bioelectromagnetics, and AI-assisted systems interpretation.
Explores ECS involvement in inflammation, autoimmunity, immune surveillance, and immune-system coordination.
Connects ECS with mitochondrial bioenergetics, ATP generation, redox control, fatigue context, and system resilience.
Research provenance record connecting GcMAF, Nagalase suppression, fibrin context, macrophage activation discussion, immune signalling, and systems-biology review boundaries.
Connects sustained CB1-dominant signalling with allostatic immune suppression and cancer-surveillance failure risk.
Research family
Metabolic and adaptive capacity records linking allostasis, mitochondria, ageing, menopause, fatigue context, metabolic reprogramming, and state-based modelling.
A four-paper series around state-based health modelling, early instability, biomarker coupling, and human-in-the-loop governance.
Frames metabolic reprogramming as a core systems-biology and oncology intelligence layer.
Connects ECS with mitochondrial bioenergetics, ATP generation, redox control, fatigue context, and system resilience.
Connects ECS signalling with ageing, hormone context, longevity, allostasis, and adaptive capacity.
Frames menopause as a systems-level regulatory transition involving hormonal withdrawal, allostatic load, and adaptive capacity.
Frames chronic THC exposure through metabolic allostasis, glycaemic dysregulation, and context-dependent biological risk.
Research family
The exploratory systems biology layer: DNA, proton tunnelling, biophotonics, bioelectromagnetic interfaces, and speculative physics boundaries.
Explores DNA systems, multidimensional interpretation, and speculative physics context within the broader systems biology estate.
Frames DNA as a bioelectromagnetic interface for cellular intelligence and information-state interpretation.
Connects proton tunnelling, base-pair instability, epigenetic collapse, and oncogenesis as a systems biology research thread.
Frames biophotonic regulation as an exploratory biological signalling layer within systems-oriented interpretation.
A speculative exploratory-perception record connecting DNA, ECS modulation, and AI-guided resonance framing.
Research family
Safety and governance records for non-claims, identity-threat, AI-use boundaries, irreversibility, constraints, and controlled public access.
Defines the QTOS capability envelope, limits, guardrails, non-claims, and infrastructure-grade scope.
Defines Rubik as a constraint-first computation and governance layer below QTOS, focused on reachability, impossibility, and deterministic boundaries.
Distinguishes descriptive mapping from constraint systems that define what is reachable, blocked, or structurally impossible.
Explores identity-threat effects in scientific reasoning, useful for safety, governance, and non-claims framing.
Frames irreversibility, intervention limits, and biological state governance as a safety and systems problem.
Positions THC as a regulatory modulator whose effects depend on biological context, indication, resilience, and system state.
Full master DOI page
This is the flat master list for quick review. Grouped sections above explain the story; this section keeps every named DOI record in one place.
Drug Resistance and Persistent Signalling States: A Compartmental Redistribution Framework
2026 · Drug resistance / signalling states
Series 2 Paper VII - THC Is a Poor Proxy for Endocannabinoid Medicine
2026 · Cannabinoid risk / ECS integrity
Series 2 Paper VIII Indication-Aware Cannabinoid Frameworks
2026 · Cannabinoid risk / ECS integrity
CB1-Dominant Signalling, Allostatic Immune Suppression, and Failure of Cancer Surveillance
2026 · Cannabinoid risk / oncology
A Systems-Level Framework for Understanding THC as a Regulatory Modulator in Complex Clinical Care
2026 · Cannabinoid risk / clinical context
Autonomic–Cardiovascular Allostasis Under Sustained CB1 Signalling
2026 · Cannabinoid risk / autonomic systems
THC, Metabolic Allostasis, and Glycaemic Dysregulation (Volume 2: Context-Dependent Risks of Chronic THC Exposure)
2026 · Cannabinoid risk / metabolism
Context-Dependent Risks of THC: Why Indication-Aware Framing Is Essential in Cannabinoid Medicine
2026 · Cannabinoid risk / ECS integrity
From Mapping Failure to Enforcing Impossibility: A Formal Distinction Between Descriptive State-Space Analysis and Constitutive Constraint Systems
2026 · Constraint systems / Rubik
Menopause as a Regulatory Phase Transition: A Systems-Level View of Hormonal Withdrawal, Allostatic Load, and Adaptive Capacity
2026 · Adaptive capacity / endocrine systems
A Systems-Level Review of Identity-Threat in Scientific Reasoning
2026 · Governance / reasoning safety
Adaptive Biological Systems & State-Based Health Modelling: A Four-Paper Research Series
2026 · Adaptive systems
Context-Dependent Cannabinoid Risk and Endocannabinoid System Integrity: Allostasis, Adaptive Capacity, and Indication-Aware Frameworks
2026 · Cannabinoid risk / ECS integrity
ECS & Cancer: Tumour Microenvironment, Immune Evasion, and Apoptosis
2026 · Oncology / ECS
Endocannabinoid System Dysregulation as a Meta-Hallmark of Cancer: A Systems-Level Failure of Biological Coordination
2026 · Oncology / ECS meta-hallmark
Hallmark-Driven Cancer Suppression via Programmed Cell Death Coordination: A Systems-Biology and Endocannabinoid Signalling Framework
2026 · Oncology / PCD / hallmarks
Causal Sovereignty: Governing Irreversibility in Biological Systems
2026 · Governance / irreversibility
Rubik vs Supercomputers: Constraint-First Computation and Governance Infrastructure
2025 · Rubik / constraint-first computation
Biophotonic Regulation as Biological Intelligence Infrastructure: A Systems-Level Framework for AI-Driven Precision Medicine
2025 · Biophotonics / systems biology
QTOS Capability Envelope & System Guardrails (v1.0): Formal Definition of Scope, Limits, and Non-Claims
2025 · QTOS / safety guardrails
A Physics-First Framework for Multi-Layer Disease Dynamics
2025 · Physics-first biology / QTOS
DNA as an Antenna: A Bioelectromagnetic Interface for Cellular Intelligence
2025 · DNA / bioelectromagnetics
Proton Tunnelling and Oncogenesis: From Base Pair Instability to Epigenetic Collapse
2025 · DNA / oncogenesis
ECS and Pain, Sleep, Sensory Modulation: A Bioelectric–Neuroimmune Framework
2025 · ECS / pain / sleep / sensory
GcMAF, Nagalase Suppression & Fibrin Reduction: A Therapeutic White Paper
2025 · GcMAF / immune signalling / microbiome-oncology context
miRNA Bio intelligence Vol 1
2025 · miRNA / cell-state intelligence
An Integrated Systems Framework for Epilepsy: From Mitochondrial Collapse to Biophotonic Modulation and AI-Driven Intervention
2025 · Neuro / epilepsy / mitochondrial systems
miRNA Neuroplasticity OS – Volume II
2025 · miRNA / neuroplasticity
A Programmable Therapeutic Operating System for Neural, Cognitive, and Gut–Brain–ECS Reprogramming
2025 · Programmable OS / neuro / gut-brain-ECS
A Therapeutic Framework Integrating 22 Hallmarks of Cancer with ECS, Bioelectric, and Systems Biology Layers
2025 · Oncology / hallmarks
Neuro Cell Integration Dataset & Framework
2025 · Neuro cells / systems integration
QTOS Systems-State Framework
2025 · QTOS / platform OS
AI-Driven Systems Immunology: Immune System Bioenergetics
2025 · Immunology / bioenergetics
ECS & Aging, Hormones, and Longevity
2025 · ECS / ageing / hormones
ECS & Immunology: Inflammation, Autoimmunity, and Immune Surveillance
2025 · ECS / immunology
ECS & Mitochondrial Bioenergetics, ATP Generation, Redox Control, and Fatigue Resolution
2025 · ECS / mitochondria / bioenergetics
Exploratory Remote-Perception Systems: DNA, ECS Modulation, and AI-Guided Resonance Framing
2025 · Exploratory perception / speculative systems
Advanced Metabolic Reprogramming
2025 · Metabolic reprogramming
AI-driven immunology, systems bioenergetics, bioelectromagnetic modulation, immunometabolism, epigenetic immunomodulation, precision immunotherapy, immune recalibration, computational biology, personalized medicine
2025 · Immunology / systems bioenergetics
ECS PCD Omni Modulation Framework
2025 · ECS / programmed cell death
Systems Oncology Treatment Framework
2025 · Systems oncology
ECS–Microbiome Integration: A Multi-Domain Systems Biology Framework
2025 · ECS / microbiome / systems biology
The Endocannabinoid System as a Systems-Regulated Operating Model: A Multidimensional Framework for Human Biology Reprogramming
2025 · ECS operating system
The OV-KillSwitch Expansion Pack: Nutrigenetic, Microbiome, Frequency, and Enzyme-Driven Precision Oncology
2025 · Oncology / OV systems
Nervous System Reprogramming: A Multi-Dimensional Framework for Neural Epigenetics, Resonance Tuning, and ECS-Modulated Precision Repair
2025 · Neuro / ECS / epigenetics
DNA & Multidimensional Systems Hypothesis
2025 · DNA / speculative physics
Public proof, protected platform
The public platform page explains the estate, engine families, proof layer, and access pathway. Deeper technical assets, app surfaces, raw datasets, and controlled demos remain protected until review.
Public layer
Protected layer
Public visibility is not a licence. Public pages do not grant access to protected material, commercial terms, AI ingestion, derivative databases, model training, redistribution, replication, or clinical use.
Reviewed access
Public DOI records support provenance and review. Protected manuscripts, source files, data-room extracts, buyer packs, internal app surfaces, and commercial terms require controlled access.
Request research review